It is highly likely that the Alzheimer’s results from a complex and interrelated combination of genetic and non-genetic factors.
These so called risk factors influence a person’s risk to developing Alzheimer’s disease. Currently, each of these risk factors is the subject of a great deal of research around the world.
Although cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes, and smoking, are associated with a higher risk of onset and course of Alzheimer’s, statins, which are cholesterol lowering drugs, have not been effective in preventing or improving the course of the disease.
Long-term usage of non-steroidal anti-inflammatory drugs (NSAIDs) is associated with a reduced likelihood of developing AD. Evidence also support the notion that NSAIDs can reduce inflammation related to amyloid plaques.
A person’s genetic make up can directly influence the chances for the onset and development of Alzheimer’s. A person’s genes are inherited from their biological parents and passed along family lines to their biological children.
There are two main types of Alzheimer’s Disease.
Familial Alzheimer’s Disease (FAD): is a very rare form of Alzheimer’s Disease which runs in families. Also known as Early Onset Alzheimer’s or Younger Onset Alzheimer’s.
Sporadic Alzheimer’s Disease: is the most common form of Alzheimer’s Disease, but researchers are still trying to work out how this develops. Also known as Late Onset Alzheimer’s.